Much of the stuff about salt and heart health is bullshit. It’s from a well publicized hypothesis that was never tested
Having said that we don’t need much, I don’t add salt to the beef which is more than 90% of my diet, I do add a small amount of salt to water after a heavy drinking session, it’s good for rehydration, and I don’t drink, especially heavily often
When I was on keto I took a lot of salt (which is why I looked up the current scientific position on it) and I can tell you it’s pretty difficult to take too much, salt tastes terrible if you have had enough
The thing that increased heart disease: same as everything else, sugar. It prevents use of the circulating fat, which is carried in high density cholesterol. Fatty acids (fats) build up as you eat more fats but cannot burn them because carbohydrates must be burnt first
The HDL cholesterol exceeds its lifespan with nowhere to deliver the fat and breaks up, this increases free fatty acids (with no protective cholesterol cage) and small fragments of the HDL cage, those fragments are the ones that embed in the walls of blood vessels, collect calcium and eventually break off as a calcified clump and blocks something important
Much of the stuff about salt and heart health is bullshit. It’s from a well publicized hypothesis that was never tested
Having said that we don’t need much, I don’t add salt to the beef which is more than 90% of my diet, I do add a small amount of salt to water after a heavy drinking session, it’s good for rehydration, and I don’t drink, especially heavily often
When I was on keto I took a lot of salt (which is why I looked up the current scientific position on it) and I can tell you it’s pretty difficult to take too much, salt tastes terrible if you have had enough
The thing that increased heart disease: same as everything else, sugar. It prevents use of the circulating fat, which is carried in high density cholesterol. Fatty acids (fats) build up as you eat more fats but cannot burn them because carbohydrates must be burnt first
The HDL cholesterol exceeds its lifespan with nowhere to deliver the fat and breaks up, this increases free fatty acids (with no protective cholesterol cage) and small fragments of the HDL cage, those fragments are the ones that embed in the walls of blood vessels, collect calcium and eventually break off as a calcified clump and blocks something important